antimonytrisulfide

This chemical does not meet GHS hazard criteria for 0.8% (3 of 359) of reports.

H302 (70.5%): Harmful if swallowed [Warning Acute toxicity, oral]

H332 (70.5%): Harmful if inhaled [Warning Acute toxicity, inhalation]

H351 (27.3%): Suspected of causing cancer [Warning Carcinogenicity]

H373 (27.3%): May causes damage to organs through prolonged or repeated exposure [Warning Specific target organ toxicity, repeated exposure]

H411 (63.8%): Toxic to aquatic life with long lasting effects [Hazardous to the aquatic environment, long-term hazard]

P203, P260, P261, P264, P270, P271, P273, P280, P301+P317, P304+P340, P317, P318, P319, P330, P391, P405, and P501 (click each P-code to see the statement)

Aggregated GHS information provided per 359 reports by companies from 16 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.

Reported as not meeting GHS hazard criteria per 3 of 359 reports by companies.

There are 15 notifications provided by 356 of 359 reports by companies with hazard statement code(s).

Information may vary between notifications depending on impurities, additives, and other factors. The percentage value in parenthesis indicates the notified classification ratio from companies that provide hazard codes. Only hazard codes with percentage values above 10% are shown. For more detailed information, please visit ECHA C&L website.

H350: May cause cancer [Danger Carcinogenicity]

H402: Harmful to aquatic life [Hazardous to the aquatic environment, acute hazard]

P203, P273, P280, P318, P405, and P501 (click each P-code to see the statement)

H319: Causes serious eye irritation [Warning Serious eye damage/eye irritation]

H372: Causes damage to organs through prolonged or repeated exposure [Danger Specific target organ toxicity, repeated exposure]

P260, P264, P264+P265, P270, P280, P305+P351+P338, P319, P337+P317, and P501 (click each P-code to see the statement)

H401: Toxic to aquatic life [Hazardous to the aquatic environment, acute hazard]

H411: Toxic to aquatic life with long lasting effects [Hazardous to the aquatic environment, long-term hazard]

P273, P391, and P501 (click each P-code to see the statement)

Suitable extinguishing media: Use water spray, alcohol-resistant foam, dry chemical or carbon dioxide.

Advice for firefighters: Wear self-contained breathing apparatus for firefighting if necessary.

ACCIDENTAL RELEASE MEASURES: Personal precautions, protective equipment and emergency procedures: Use personal protective equipment. Avoid dust formation. Avoid breathing vapors, mist or gas. Ensure adequate ventilation. Avoid breathing dust. Environmental precautions Prevent further leakage or spillage if safe to do so. Do not let product enter drains. Discharge into the environment must be avoided. Methods and materials for containment and cleaning up: Pick up and arrange disposal without creating dust. Sweep up and shovel. Keep in suitable, closed containers for disposal.

SRP: Recycle any unused portion of the material for its approved use or return it to the manufacturer or supplier. Ultimate disposal of the chemical must consider: the material's impact on air quality; potential migration in air, soil or water; effects on animal, aquatic and plant life; and conformance with environmental and public health regulations. If it is possible or reasonable use an alternative chemical product with less inherent propensity for occupational harm/injury/toxicity or environmental contamination.

Product: Offer surplus and non-recyclable solutions to a licensed disposal company; Contaminated packaging: Dispose of as unused product.

ACCIDENTAL RELEASE MEASURES: Personal precautions, protective equipment and emergency procedures: Use personal protective equipment. Avoid dust formation. Avoid breathing vapors, mist or gas. Ensure adequate ventilation. Avoid breathing dust. Environmental precautions Prevent further leakage or spillage if safe to do so. Do not let product enter drains. Discharge into the environment must be avoided.

Precautions for safe handling: Further processing of solid materials may result in the formation of combustible dusts. The potential for combustible dust formation should be taken into consideration before additional processing occurs. Provide appropriate exhaust ventilation at places where dust is formed.

Appropriate engineering controls: Handle in accordance with good industrial hygiene and safety practice. Wash hands before breaks and at the end of workday.

Gloves must be inspected prior to use. Use proper glove removal technique (without touching glove's outer surface) to avoid skin contact with this product. Dispose of contaminated gloves after use in accordance with applicable laws and good laboratory practices. Wash and dry hands.

For more Preventive Measures (Complete) data for Antimony trisulfide (8 total), please visit the HSDB record page.

Keep container tightly closed in a dry and well-ventilated place. Keep in a dry place. Keep in a dry place. Storage class (TRGS 510): Non-combustible, acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects.

0.5 [mg/m3], as Sb

50.0 [mg/m3], as Sb

50 mg/cu m, as Sb /Antimony/

8 hr Time Weighted Avg (TWA): 0.5 mg/cu m. /Antimony and compounds, as Sb/

Peak Exposure Recommendation: Transient increases in workers' exposure levels may exceed 3 times the value of the TLV-TWA level for no more than 15 minutes at a time, on no more than 4 occasions spaced 1 hour apart during a workday, and under no circumstances should they exceed 5 times the value of the TLV-TWA level. In addition, the 8-hour TWA is not to be exceeded for an 8-hour work period. /Antimony and compounds, as Sb/

Eye/face protection: Use equipment for eye protection tested and approved under appropriate government standards such as NIOSH (US) or EN 166(EU).

Skin protection: Handle with gloves.

Body Protection: Choose body protection in relation to its type, to the concentration and amount of dangerous substances, and to the specific work-place.

Respiratory protection: Respiratory protection is not required. Where protection from nuisance levels of dusts are desired, use type N95 (US) or type P1 (EN 143) dust masks. Use respirators and components tested and approved under appropriate government standards such as NIOSH (US) or CEN (EU).

For more Personal Protective Equipment (PPE) (Complete) data for Antimony trisulfide (12 total), please visit the HSDB record page.

CBI; Other Solid; Dry Powder; Dry Powder; Large Crystals

Black solid (naturally occurring form, stibnite) or orange-red solid; [Hawley] If the unstable red-orange modification occurs along with the black, crystalline form, the mixture can be carmine or brownish red; [HSDB] Black odorless chunks; [MSDSonline]

Gray, lustrous, crystalline masses or grayish-black powder; also exists in red modification

Gray-black orthogonal crystals

Black crystals; orange-red crystals

Red to black crystals

Has an unstable red-orange modification. If this occurs along with the black, crystalline form, the mixture can be carmine or brownish red.

Insoluble in water

Practically insoluble in water

In water, 0.002/100 at 20 °C (decomposes)

Soluble in concentrated hydrochloric acid

For more Solubility (Complete) data for Antimony trisulfide (7 total), please visit the HSDB record page.

4.562 g/cu cm at 25 °C

Stable under recommended storage conditions.

Hazardous decomposition products formed under fire conditions - Antimony oxide.

When heated to decomposition ... it emits highly toxic fumes of oxides of sulfur and antimony.

NATURAL STIBNITE: INDEX OF REFRACTION: 3.194, 4.064, 4.303.

Black crystals - occurs in nature as black-cyrstalline stibnite; orange-red crystals - as precipitated from solutions of salt of antimony, the trisulfide is an orange-red precipitate, which is filtered, dried and ground

When the crystalline form of antimony trisulfide is heated in air, it burns with a blue flame.

The crystal structure of stibnite contains two distinctly different antimony sites and consists of two parallel antimony trisulfide (Sb4S6) chains that are linked together to form crumpled sheets (two per unit cell).

In the absence of air, antimony trisulfide dissolves in alkaline sulfide solutions to form the thioantimonate(III) ion; in the presence of air the tetrathioantimonate(V) ion is formed.

For more Other Experimental Properties (Complete) data for Antimony trisulfide (8 total), please visit the HSDB record page.

Metals -> Metalloid Compounds (Antimony)

Incompatible materials: Strong oxidizing agents. Do not store near acids.

Spontaneously flammable when exposed to strong oxidizers. ... Moderately explosive by spontaneous reaction with chlorates, perchlorates, /chlorine monoxide/, thallic oxide. ... On contact with acid or acid fumes it emits highly toxic fumes of oxides of sulfur and antimony. Will react with water or steam to produce toxic and flammable vapors.

... Chlorine monoxide ... causes explosions with sulfides of barium, tin, antimony and mercury.

The reaction at ordinary temperatures between fluorine and antimony trisulfide is accompanied by a blue flame.

For more Hazardous Reactivities and Incompatibilities (Complete) data for Antimony trisulfide (12 total), please visit the HSDB record page.

IDENTIFICATION AND USE: Antimony trisulfide is a solid, grey or red-orange. It is used in pyrotechnics, as well as in the manufacture of ruby glass, matches, explosives, and as a pigment in paints. HUMAN STUDIES: Workmen in a plant where antimony trisulfide was used were studied, focusing on cardiovascular status. Prior to the study there had been six sudden deaths in a group of 125 workers exposed to antimony for 8 months to 2 years. The deaths were suspected to be due to heart disease. In the workers studied, 14 of 113 had blood pressures that were >140/90 mm Hg, and 37 of 75 showed significant changes in their EKGs, mostly the T-wave. Ulcers were detected in 7 of 111 exposed persons (63 per 1,000) as compared with 15 per 1,000 in the total plant population. Although use of antimony trisulfide was discontinued, EKG changes persisted in 12 of 665 workers. ANIMAL STUDIES: Degenerative changes in the liver and in the tubular epithelium of the kidney were observed in rabbits exposed to 27.8 mg/cu m antimony trisulfide for five days. Rats exposed to 3.1 mg/cu m antimony trisulfide by inhalation for six weeks developed electrocardiographic changes, notably with flattened T-waves. On autopsy, the heart was found to be dilated, with signs of degenerative changes; focal hemorrhage and congestion in the lungs were considered to be secondary to heart failure. Cardiotoxic changes were observed in two dogs exposed to 5.6 mg/cu m for ten weeks. Continuous oral administration of antimony for 8 and 12 weeks had toxic effect on the structure and function of the kidney in growing rat.

The inhalation data suggest that the myocardium is a target of antimony toxicity. It is possible that antimony affects circulating glucose by interfering with enzymes of the glycogenolysis and gluconeogenesis pathways. The mechanism of action of antimony remains unclear. However, some studies suggest that antimony combines with sulfhydryl groups including those in several enzymes important for tissue respiration. The antidotal action of BAL depends on its ability to prevent or break the union between antimony and vital enzymes. Moreover, the The cause of death is believed to be essentially the same as that in acute arsenic poisoning. (T18, L741, A238)

Evaluation: There is inadequate evidence for the carcinogenicity of antimony trisulfide in humans. There is limited evidence for the carcinogenicity of antimony trisulfide in experimental animals. Overall evaluations: Antimony trisulfide is not classifiable as to its carcinogenicity to humans (Group 3).

3, not classifiable as to its carcinogenicity to humans. (L135)

Dermal exposure to antimony can cause antimony spots (papules and pustules around sweat and sebaceous glands). Antimony poisoning can also lead to pneumoconiosis. Alterations in pulmonary function and other effects including chronic bronchitis, chronic emphysema, inactive tuberculosis, pleural adhesions, and irritation can result from inhalation of antimony. Increased blood pressure can also result from antimony poisoning. Myocardial depression, vasodilation and fluid loss may cause shock with hypotension, electrolyte disturbances and acute renal failure. Cerebral oedema, coma, convulsions, and death are possible. (L741)

Inhalation (L741) ; oral (L741) ; dermal (L741

Abdominal pain, vomiting, diarrhea can result from inhalation of antimony. Dyspnea, headache, vomiting,cough, conjunctivitis, and bloody purulent discharge from nose can result from inhalation exposure. Skin or eye contact can cause pain and redness of the exposed surface. (T64, L741)

Occupational hepatotoxin - Secondary hepatotoxins: the potential for toxic effect in the occupational setting is based on cases of poisoning by human ingestion or animal experimentation.

IARC Carcinogen - Class 2: International Agency for Research on Cancer classifies chemicals as probable (2a), or possible (2b) human carcinogens.

LD50: 20 000 mg/kg (Oral, Rat) (L742)

LD50 Rat ip 1 g/kg

LD50 Mouse ip 209 mg/kg

Following oral exposure to antimony, administer charcoal as a slurry (240 mL water/30 g charcoal). Following inhalation exposure, move patient to fresh air. Monitor for respiratory distress. If cough or difficulty breathing develops, evaluate for respiratory tract irritation, bronchitis, or pneumonitis. Administer oxygen and assist ventilation as required. Treat bronchospasm with inhaled beta2 agonist and oral or parenteral corticosteroids. In case of eye exposure, irrigate exposed eyes with copious amounts of room temperature water for at least 15 minutes. Following dermal exposure, remove contaminated clothing and wash exposed area thoroughly with soap and water. A physician may need to examine the area if irritation or pain persists. (T36)

/SRP:/ Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR if necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on the left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Poisons A and B/

/SRP:/ Basic treatment: Establish a patent airway (oropharyngeal or nasopharyngeal airway, if needed). Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations if needed. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for pulmonary edema and treat if necessary ... . Monitor for shock and treat if necessary ... . Anticipate seizures and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with 0.9% saline (NS) during transport ... . Do not use emetics. For ingestion, rinse mouth and administer 5 mL/kg up to 200 mL of water for dilution if the patient can swallow, has a strong gag reflex, and does not drool ... . /Poisons A and B/

/SRP:/ Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious, has severe pulmonary edema, or is in severe respiratory distress. Positive-pressure ventilation techniques with a bag valve mask device may be beneficial. Consider drug therapy for pulmonary edema ... . Consider administering a beta agonist such as albuterol for severe bronchospasm ... . Monitor cardiac rhythm and treat arrhythmias as necessary ... . Start IV administration of D5W TKO /SRP: "To keep open", minimal flow rate/. Use 0.9% saline (NS) or lactated Ringer's (LR) if signs of hypovolemia are present. For hypotension with signs of hypovolemia, administer fluid cautiously. Watch for signs of fluid overload ... . Treat seizures with diazepam or lorazepam ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Poisons A and B/

/SRP:/ Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand-valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR as necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Hydrogen Sulfide and Related Compounds/

For more Antidote and Emergency Treatment (Complete) data for Antimony trisulfide (6 total), please visit the HSDB record page.

/EPIDEMIOLOGY STUDIES/ Workmen in a plant where antimony trisulfide was used in the manufacture of grinding wheels were studied. This study focused on cardiovascular status, since prior to the study there had been six sudden deaths in a group of 125 workers exposed to antimony for 8 months to 2 years. The deaths were suspected to be due to heart disease. In the workers studied, 14 of 113 had blood pressures that were >140/90 mm Hg, and 37 of 75 showed significant changes in their EKGs, mostly the T-wave. Ulcers were detected in 7 of 111 exposed persons (63 per 1,000) as compared with 15 per 1,000 in the total plant population. No other disorders suspected of being related to antimony exposure were observed. Although use of antimony trisulfide was discontinued, EKG changes persisted in 12 of 665 workers subsequently reexamined.

/LABORATORY ANIMALS: Acute Exposure/ Degenerative changes in the liver and in the tubular epithelium of the kidney were observed in rabbits exposed to 27.8 mg/cu m antimony trisulfide for five days.

/LABORATORY ANIMALS: Subchronic or Prechronic Exposure/ Rats exposed to 3.1 mg/cu m antimony trisulfide by inhalation for six weeks developed electrocardiographic changes, notably with flattened T-waves; on autopsy, the heart was found to be dilated, with signs of degenerative changes; focal hemorrhage and congestion in the lungs were considered to be secondary to heart failure. Similar pathological effects were seen in rabbits exposed to 5.6 mg/cu m for six weeks.

/LABORATORY ANIMALS: Subchronic or Prechronic Exposure/ Cardiotoxic changes were observed in two dogs exposed to 5.6 mg/cu m for ten weeks, but not in two dogs exposed to 5.3 mg/cu m for seven weeks.

/LABORATORY ANIMALS: Subchronic or Prechronic Exposure/ Contamination of the environment with antimony compounds may affect human health through the persistent exposure to small doses over a long period. Sixty growing male albino rats, weighing 43-57 grams, were utilized in this study. The animals were divided into 3 groups; each of 20 rats: animals of group I served as control, animals of group II received 6 mg/kg body weight antimony trisulfide daily for 8 weeks with drinking water, and those of group III received the same dose by the same route for 12 weeks. The Malpighian renal corpuscles showed distortion, destruction and congestion of glomerular tuft, vacuoles in the glomeruli, peritubular hemorrhage, obliteration of Bowman's space, and thickening with irregularity of Bowman's membrane. The proximal convoluted tubules demonstrated patchy loss of their brush border, thickening of the basement membrane with loss of its basal infoldings, disarrangement of the mitochondria, pleomorphic vacuoles in the cytoplasm, apical destruction of the cells, apical migration of the nuclei, and absence of microvilli. On the other hand, peri-tubular hemorrhage, apical vacuolation, small atrophic nuclei, swelling of mitochondria, obliteration of the lumina, destruction of cells, and presence of tissue debris in the lumina, were observed in the distal convoluted tubules. The present work demonstrated the hazardous effect of antimony on the renal function as evidenced by the significant increase of the level of blood urea, serum creatinine, and serum sodium and potassium. In conclusion, this study proposed that continuous oral administration of antimony for 8 and 12 weeks has hazardous toxic effect on the structure and function of the kidney in growing albino rat. Based on the results of the present study, it is recommended to avoid the use of any drinking water contaminated with antimony compounds and forbidden its use in infants and children foods.

Antimony is naturally present in the earth's crust and is released to the environment in windblown dust, from volcanic eruptions, sea spray, forest fires, and biogenic sources(1). It is estimated that 41% of the antimony emitted to the environment is from natural sources(1). The median percentage contribution of these sources are estimated to be: wind-borne soil particles, 32.5%; volcanoes, 29.6%; sea salt spray, 23.3%; forest fires, 9.2%; and biogenic sources, 12.1%(1). While not an abundant element, antimony occurs in over 100 minerals, the more commercially important of which are sulfides and, to a lesser extent, oxides of Sb(III)(2,3). Elemental antimony very seldom occurs in nature due to its high attraction for sulfur and metallic elements such as copper, lead and silver(4). Atimony is frequently found in deposits of other metals such as lead, copper, and silver(2-3). Antimony ore is mined in China, Mexico, and Bolivia(5).

The few results from bioconcentration studies as well as the levels of antimony found in aquatic species suggest that antimony does not bioconcentrate in fish(1,2). Bioconcentration factors reported for antimony range from 2-9(1). No detectable amount of bioconcentration of antimony occurred in a 28-day test with bluegills(3). An exception is a BCF of 16,000 reported for invertebrates(2). Results of a study of the distribution of antimony around a smelter suggest that antimony does not biomagnify in the food chain(2).

Antimony is naturally present in the earth's crust and is released to the environment in windblown dust, from volcanic eruptions, sea spray, forest fires, and biogenic sources(1). It is estimated that 41% of the antimony emitted to the environment is from natural sources(1). The median percentage contribution of these sources are estimated to be: wind-borne soil particles, 32.5%; volcanoes, 29.6%; sea salt spray, 23.3%; forest fires, 9.2%; and biogenic sources, 12.1%(1). While not an abundant element, antimony occurs in over 100 minerals, the more commercially important of which are sulfides and, to a lesser extent, oxides of Sb(III)(2,3). Elemental antimony very seldom occurs in nature due to its high attraction for sulfur and metallic elements such as copper, lead and silver(4). Atimony is frequently found in deposits of other metals such as lead, copper, and silver(2-3). Antimony ore is mined in China, Mexico, and Bolivia(5).

The few results from bioconcentration studies as well as the levels of antimony found in aquatic species suggest that antimony does not bioconcentrate in fish(1,2). Bioconcentration factors reported for antimony range from 2-9(1). No detectable amount of bioconcentration of antimony occurred in a 28-day test with bluegills(3). An exception is a BCF of 16,000 reported for invertebrates(2). Results of a study of the distribution of antimony around a smelter suggest that antimony does not biomagnify in the food chain(2).

SRP: Recycle any unused portion of the material for its approved use or return it to the manufacturer or supplier. Ultimate disposal of the chemical must consider: the material's impact on air quality; potential migration in air, soil or water; effects on animal, aquatic and plant life; and conformance with environmental and public health regulations. If it is possible or reasonable use an alternative chemical product with less inherent propensity for occupational harm/injury/toxicity or environmental contamination.

Product: Offer surplus and non-recyclable solutions to a licensed disposal company; Contaminated packaging: Dispose of as unused product.

/GUIDE 157 SUBSTANCES - TOXIC and/or CORROSIVE (Non-Combustible/Water-Sensitive)/ Fire or Explosion: Non-combustible, substance itself does not burn but may decompose upon heating to produce corrosive and/or toxic fumes. For UN1796, UN1826, UN2031 at high concentrations and for UN2032, these may act as oxidizers, also consult GUIDE 140. Vapors may accumulate in confined areas (basement, tanks, hopper/tank cars, etc.). Substance may react with water (some violently), releasing corrosive and/or toxic gases and runoff. Contact with metals may evolve flammable hydrogen gas. Containers may explode when heated or if contaminated with water. /Antimony compound, inorganic, solid, n.o.s.; Antimony compound, inorganic, liquid, n.o.s./

/GUIDE 157 SUBSTANCES - TOXIC and/or CORROSIVE (Non-Combustible/Water-Sensitive)/ Health: TOXIC; inhalation, ingestion or contact (skin, eyes) with vapors, dusts or substance may cause severe injury, burns or death. Reaction with water or moist air may release toxic, corrosive or flammable gases. Reaction with water may generate much heat that will increase the concentration of fumes in the air. Fire will produce irritating, corrosive and/or toxic gases. Runoff from fire control or dilution water may be corrosive and/or toxic and cause pollution. /Antimony compound, inorganic, solid, n.o.s.; Antimony compound, inorganic, liquid, n.o.s./

/GUIDE 157 SUBSTANCES - TOXIC and/or CORROSIVE (Non-Combustible/Water-Sensitive)/ Public Safety: CALL Emergency Response Telephone Number on Shipping Paper first. If Shipping Paper not available or no answer, refer to appropriate telephone number listed on the inside back cover. As an immediate precautionary measure, isolate spill or leak area in all directions for at least 50 meters (150 feet) for liquids and at least 25 meters (75 feet) for solids. Keep unauthorized personnel away. Stay upwind, uphill and/or upstream. Ventilate enclosed areas. /Antimony compound, inorganic, solid, n.o.s.; Antimony compound, inorganic, liquid, n.o.s./

/GUIDE 157 SUBSTANCES - TOXIC and/or CORROSIVE (Non-Combustible/Water-Sensitive)/ Protective Clothing: Wear positive pressure self-contained breathing apparatus (SCBA). Wear chemical protective clothing that is specifically recommended by the manufacturer. It may provide little or no thermal protection. Structural firefighters' protective clothing provides limited protection in fire situations ONLY; it is not effective in spill situations where direct contact with the substance is possible. /Antimony compound, inorganic, solid, n.o.s.; Antimony compound, inorganic, liquid, n.o.s./

For more DOT Emergency Guidelines (Complete) data for Antimony trisulfide (8 total), please visit the HSDB record page.

UN 3141; Antimony compound, inorganic, liquid, not otherwise specified

UN 1549; Antimony compound, inorganic, solid, not otherwise specified

IMO 6.1; Antimony compound, inorganic, solid, not otherwise specified; Antimony compound, inorganic, liquid, not otherwise specified

49 411 15; Antimony sulfide, solid

No person may /transport,/ offer or accept a hazardous material for transportation in commerce unless that person is registered in conformance ... and the hazardous material is properly classed, described, packaged, marked, labeled, and in condition for shipment as required or authorized by ... /the hazardous materials regulations (49 CFR 171-177)./

The International Air Transport Association (IATA) Dangerous Goods Regulations are published by the IATA Dangerous Goods Board pursuant to IATA Resolutions 618 and 619 and constitute a manual of industry carrier regulations to be followed by all IATA Member airlines when transporting hazardous materials. Antimony compound, inorganic, solid, not otherwise specified and antimony compound, inorganic, liquid, not otherwise specified are included on the dangerous goods list. /Antimony compound, inorganic, solid, not otherwise specified; Antimony compound, inorganic, liquid, not otherwise specified/

The International Maritime Dangerous Goods Code lays down basic principles for transporting hazardous chemicals. Detailed recommendations for individual substances and a number of recommendations for good practice are included in the classes dealing with such substances. A general index of technical names has also been compiled. This index should always be consulted when attempting to locate the appropriate procedures to be used when shipping any substance or article. Antimony compound, inorganic, solid, not otherwise specified and antimony compound, inorganic, liquid, not otherwise specified are included on the dangerous goods list. /Antimony compound, inorganic, solid, not otherwise specified; Antimony compound, inorganic, liquid, not otherwise specified/